Please use this identifier to cite or link to this item: https://scidar.kg.ac.rs/handle/123456789/10163
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dc.contributor.authorRadosavljevic, Gordana-
dc.contributor.authorJovanovic I.-
dc.contributor.authorKnezevic M.-
dc.contributor.authorZdravkovic, Nemanja-
dc.contributor.authorPavlovic S.-
dc.contributor.authorLukic, Miodrag-
dc.contributor.authorArsenijevic, Nebojsa-
dc.date.accessioned2021-04-20T15:02:32Z-
dc.date.available2021-04-20T15:02:32Z-
dc.date.issued2010-09-20-
dc.identifier.issn00271314-
dc.identifier.urihttps://scidar.kg.ac.rs/handle/123456789/10163-
dc.description.abstractWe used metastatic variant of B16 melanoma (B16F1) to study lung colonization galectin-3-deficient (gal-3-/-) C57BL/6 mice. In vivo study showed that compared with gal-3+/+ mice, gal-3-/-mice exhibited resistance to lung colonization of B16F1 melanoma cells (p < 0.03). In vitro assays showed higher number of attached malignant cells in the tissue section derived from gal-3+/+ mice (p < 0.001) and tumor specific cytotoxicity of lymphoid cells of tumour inoculated gal-3-/- suggesting that galectin-3 is considered as therapeutic target. © 2010 Allerton Press, Inc.-
dc.relation.ispartofMoscow University Chemistry Bulletin-
dc.rightsrestrictedAccess-
dc.titleRole of Galectin-3 in Tumour Metastasis-
dc.typearticle-
dc.identifier.doi10.3103/S0027131410030107-
dc.identifier.scopus77956608737-
Appears in Collections:Faculty of Medical Sciences, Kragujevac

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