Please use this identifier to cite or link to this item: https://scidar.kg.ac.rs/handle/123456789/9569
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dc.rights.licenseBY-NC-ND-
dc.contributor.authorJovanovic I.-
dc.contributor.authorPejnović, Nada-
dc.contributor.authorRadosavljevic, Gordana-
dc.contributor.authorArsenijevic, Nebojsa-
dc.contributor.authorLukic, Miodrag-
dc.date.accessioned2020-09-19T18:36:20Z-
dc.date.available2020-09-19T18:36:20Z-
dc.date.issued2012-
dc.identifier.issn2162-4011-
dc.identifier.urihttps://scidar.kg.ac.rs/handle/123456789/9569-
dc.description.abstractInterleukin-33, a ligand for ST2/T1, has an important role in allergy, autoimmunity and inflammation. The role of IL-33/ST2 axis in cancer is not elucidated. Using metastatic breast cancer model we provide evidence that lack of ST2 signaling led to reduced tumor growth and metastasis and enhanced anti-tumor immunity. © 2012 Landes Bioscience.-
dc.rightsopenAccess-
dc.rights.urihttps://creativecommons.org/licenses/by-nc-nd/4.0/-
dc.sourceOncoImmunology-
dc.titleIL-33/ST2 axis in innate and acquired immunity to tumors-
dc.typecontributionToPeriodical-
dc.identifier.doi10.4161/onci.1.2.18131-
dc.identifier.scopus2-s2.0-84859924039-
Appears in Collections:Faculty of Medical Sciences, Kragujevac

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